Since Epitalon has been available to the general public, there have been several testimonials of healed cancer after using Epitalon. (read more on Epitalon!)
Even people with terminal cancer have been cured of their cancer, using doses ranging from 8mg to 50mg per day. There are no recommended dose as each case is different and each individual will react differently. We suggest you consult a medical professionnal for advice.
Here are 2 numbers that will help you realise how important the Cancer Business is, and why they don’t want us to get cured:
– Costs of cancer totaled 264 billion USD, in 2010 in USA, source Research America
– Costs of cancer totaled 126 billion Euros, in 2009 in European Union, source The Lancet
Here is a review of scientific studies that have already been published regarding the effectiveness of Epitalon against cancer and cancerous cells.
There are already several scientific books, and more than 20 studies that have been published on PubMed alone, answering the keywords epithalon-cancer.
Here are Three Excerpts on this Subject:
– Pineal Gland and Cancer — An Epigenetic Approach to the Control of Malignancy: Evaluation of the Role of Melatonin.
Excerpts: The secretion of the pineal hormone melatonin is under control of the hypothalamic suprachiasmatic nuclei, the seat of the central circadian clock, and conveys information concerning time of day as well as season to practically all parts of the body. This means that melatonin is an integral part of the circadian time-keeping system.
According to the summarized findings a link exists between the pineal gland and cancer, a mutual and dynamic interaction between the secretion of melatonin and malignant growth. A fresh tumor is “sensed” by the pineal gland via neuroimmunoendocrine changes leading to a stimulation of melatonin secretion which in turn activates endogenous defence processes. At this stage of cancer development melatonin can exert a direct tumor-inhibitory activity.
Note: You can find 174 studies cited at the end of this publication. Epitalon decalcifies and stimulates the pineal gland, allowing it to produce melatonine, that’s one of the mechanisms that make it successful against cancer, and that’s why people taking Epitalon can recover their deep sleep pattern, an essential factor in body immunity and repair.
– Book: Pineal Gland and Cancer — Neuroimmunoendocrine Mechanisms in Malignancy. 2011
Excerpts: The link between the pineal gland and cancer is a rapidly emerging research field due to promising experimental and clinical trials with melatonin. The pineal gland acts as a transducer of environmental light to regulate rhythmic processes, including reproductive function in seasonally breeding animals and the entrainment of circadian rhythms, such as the sleep-wake cycle, in man. This book elucidates the physiological significance of the pineal gland and surveys phenomena and mechanisms of pineal – tumor interaction at the neuroendocrine, neuroimmune, neural, and molecular levels.
Three years ago, most authors contributing to this book gathered at the Heinrich Fabri Insitute of the University of Tubingen at Blaubeuren near Ulm in Germany for the third conference on “Pineal Gland and Cancer”.
It was in Vienna during the 1930s and 1940s that W.Bergmann and P.Engel demonstrated that pineal extracts possess growth inhibitory properties on experimental rodent tumors and R.Hofstatter reported favorable results when these extracts were given to cancer patients.
Initial clinical applications of the pineal hormone for incurable cancers raise hopes for a promising future use, particularly when combined with other therapies (e.g., interleukin-2) increasing their efficacy and reducing their toxicity.
We hope that this book will help those working in this field to link the pineal gland and cancer and encourage others from diverse areas to join and thus enrich this field of research. The interplay between the pineal gland and malignancy has to be viewed in the chronobiologic framework of the largely epigenetic, highly complex, and non-lineary organized psychoneuroimmunoendocrine network as well as progressing genomic aberrations within the tumor.
Note: This group of research seems unfortunately to be mostly focussed on the role of Melatonin, the pineal hormone, but discuss also some publications about Epithalamin from page 294-303. My opinion is that they are trying to “drown the fish”, meaning that they focus their research on Melatonin, a very complex subject, instead of going right to the point, taking the shortcut to curing cancer that is much faster with the use of Epitalon!
As usual in modern science and medical research, the but is to find new ways to treat the symptoms and multiply the number of drugs to take, instead of curing the source of the problem… The pineal gland produces much more than just melatonin. They should have called their book “Melatonin and cancer”!
– Book: Experimental Studies of the Pineal Gland Preparation Epithalamin. 2001
Abstract: Twenty-five years of study have shown a wide spectrum of high biological activity of the pineal peptide preparation epithalamin. Long-term exposure to epithalamin was followed by an increase in the mean and maximum life spans and slower rates of aging of rats, mice, and D. melanogaster. Epithalamin (Epitalon) increases pineal synthesis of serotonin, N-acetylserotonin, and melatonin and night pineal secretion of melatonin in adult and old rats. The pineal preparation decreases the luteinizing hormone and prolactin levels in adult male rats as well as the threshold of the hypothalamopituitary complex to feedback inhibition by estrogens in old female rats; it slows dawn age-related cessation of estrous function in rats and induces the recurrence of estrous cydes and fertility in old, persistently estrous rats.
Epithalamin increases the levels of triiodothyronine and decreases thyroxine in serum of adult rats. It further decreases the levels of corticosterone in the serum of mice and increases the susceptibility of the hypothalamo-pituitary complex to the homeostatic inhibition of adrenocorticotropic function by glucocorticoids in old rats.
Serum insulin and triglyceride levels in rabbits are decreased by epithalamin and the tolerance to glucose and diuresis are increased.
With respect to immune function, it was found that T and B cell-mediated immunity in adult and old mice as well as the titer of thymic serum factor and the titer of thymosin-like compounds in old mice are stimulated by the pineal peptide preparation in the same way as the colony-forming activity of splenocytes in pinealectomized rats.
Epithalamin inhibits spontaneous and induced carcinogenesis and is a potent antioxidant, decreasing lipid peroxidation and stimulating the activity of CuZn superoxide dismutase.
The obtained results demonstrate a high efficiency of epithalamin therapy for prophylaxis of age-related pathology, including cancer, showing a new physiological way to slow down pathological processes and to extend human life spans.
Note: epithalamin and epitalon are equivalent, epitalon being the non-animal tetrapeptide version.
We will look further at more Scientific Books and Studies in future articles. The exciting discoveries of Epitalon and health are continuing to show it’s ability to help our fight against disease and to live Longer, Healthier lives.
You don’t have to wait to discover how Epitalon could be helping you, and those you love. This is something you can do for yourself, Today!
Experience the use of Epitalon for yourself, and Order Epitalon Now, from OceansLab.
-1. Biological stability evaluation of the a2B1 receptor imaging agents: diamsar and DOTA conjugated DGEA peptide.
Huang CW, Li Z, Cai H, Shahinian T, Conti PS. Bioconjug Chem. 2011 Feb 16;22(2):256-63. doi: 10.1021/bc100388g. Epub 2011 Jan 18.
-2. [The use of peptide bioregulators for cancer prevention: results of 35 years of research experience and perspectives].
Anisimov VN, Khavinson VKh. Vopr Onkol. 2009;55(3):291-304. Review. Russian. No abstract available.
-3. Geroprotective effect of ala-glu-asp-gly peptide in male rats exposed to different illumination regimens.
Vinogradova IA, Bukalev AV, Zabezhinski MA, Semenchenko AV, Khavinson VKh, Anisimov VN. Bull Exp Biol Med. 2008 Apr;145(4):472-7.
-4. Effect of Ala-Glu-Asp-Gly peptide on life span and development of spontaneous tumors in female rats exposed to different illumination regimes.
Vinogradova IA, Bukalev AV, Zabezhinski MA, Semenchenko AV, Khavinson VKh, Anisimov VN. Bull Exp Biol Med. 2007 Dec;144(6):825-30.
-5. [The effect of heavy metal ions and peptide bioregulators on the expression of chromosome fragile sites in the individuals of different age groups and breast cancer patients].
Dzhokhadze TA, Ganozishvili MN, Lezhava TA. Georgian Med News. 2008 Sep;(162):11-4. Russian.
-6. Effect of the synthetic pineal peptide epitalon on spontaneous carcinogenesis in female C3H/He mice.
Kossoy G, Anisimov VN, Ben-Hur H, Kossoy N, Zusman I. In Vivo. 2006 Mar-Apr;20(2):253-7.
PMID: 16634527 – Free Article
-7. [Effect of epitalon and melatonin on life span and spontaneous carcinogenesis in senescence accelerated mice (SAM)].
Anisimov VN, Popovich IG, Zabezhinskii MA, Rozenfel’d SV, Khavinson VKh, Semenchenko AV, Iashin AI. Vopr Onkol. 2005;51(1):93-8. Russian.
-8.[The influence of substances revealing geroprotective of spontaneous carcinogenesis in mice].
Popovich IG. Adv Gerontol. 2004;14:105-13. Review. Russian.